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Creationists often argue against evolution by noting that we cannot observe evolution occurring on a grand scale today. In response, evolutionary scientists like to point to bacteria.
Many scientists argue that evolution is happening all the time in bacteria. Bacteria, with their brief life cycles and their ability to reproduce vast multitudes of generations within a nice, short, observable time frame, give scientists a chance to demonstrate "evolution in a Petri dish". The ability of bacteria to develop resistance to antibiotics has been trumpeted as evidence of the driving force of evolution and the ability of gene swapping and mutations to make these organisms better able to survive.
However, while bacterial resistance to antibiotics is a reality, it falls far short of demonstrating the theory that all things descended from single-celled organisms billions of years ago. In fact, bacteria that become resistant to antibiotics often do so at the cost of their "relative fitness" and can lose pre-existing cellular functions.
Bacteria develop resistance to antibiotics in several ways:
Natural resistance:
Bacteria already naturally have some degree of protection against antibiotics, which they need when they run into these enemies, like penicillin, out in the great big world. This resistance only goes so far, and most bacteria will be killed off when faced with large doses of antibiotics for a significant period of time. The bacteria with the greatest resistance ability sometimes survive, though, going on to reproduce and make a plethora of antibiotic-resistant offspring. This is why doctors warn patients to take their entire antibiotics prescription and not stop halfway after the symptoms go away. Failing to take the entire course can allow the strongest bacteria to stick around and reproduce, paving the way for the superbugs we see today.
Of course, the resistance is already present in the bacterial gene pool. While these super strong bacteria offer a basic survival-of-the-fittest demonstration, their resistance to antibiotics is not an essentially new development and therefore doesn't prove evolution in a grander sense.
Horizontal gene transfer.
Bacteria have a tremendous ability to swap genes with each other. This is vital for the health of bacteria, since they reproduce by binary fission (dividing into two parts) and do not benefit from the recombination of genes found in sexual reproduction. Antibiotic-resistant bacteria can exchange their genes with other bacteria, and thus pass on the ability to thumb their bacterial noses at modern medicine. Once again, the resistance is already there in the bacterial gene pool and is not an essentially new development.
Mutations. Mutations occur in bacteria in a variety of ways, including copy errors in the bacterial DNA and exposure to mutagens (chemicals or ionizing radiation) that affect bacteria's genetic material. Mutations have also enabled bacteria to resist antibiotics or chemical cleansers in some interesting, but not necessarily truly beneficial, ways.
For instance, some bacteria naturally produce the enzyme penicillinase, which they use to inactivate penicillin when they run into it in nature. If a bacterium has a problem with the gene that codes for shutting off the production of penicillinase, that bacterium will just keep producing the enzyme. This is great for the bacterium in the presence of a penicillin-based antibiotic regimen; in a human body filled with penicillin, this bacterium can survive to reproduce while the normal bacteria around it die. In normal life, though, the bacterium has a problem. It's putting a lot of energy into producing penicillinase, and because it can't turn the valve off, so to speak, it will have trouble doing all the other things it needs to do and will eventually penicillinase-produce itself to death.
Many bacteria develop resistance to antibiotics because something has gone wrong and they simply are not functioning properly. The loss of regulatory proteins is a big one. Some bacteria also lose full functioning in transport proteins. Transport proteins are necessary for bringing certain items into the cell. Bacteria that are resistant to Kanamycin get that way because they aren't correctly producing a transport protein, and therefore the Kanamycin can't get through the cell membrane into the bacterial cell to destroy it. If a transport protein is not functioning right, that means something is wrong with the cell, even if that lack of function does protect the bacteria from Kanamycin.
In short, broken genes can help bacteria survive in some circumstances, but we always find they do so at the expense of the general health of the bacteria. In a normal environment, these bacteria die off much more quickly than their normal, healthy relatives.
Gaining An Ability? Citrate in E coli:
In 2008, evolutionary biologist Richard Lenski of Michigan State University in East Lansing found that a population of E coli, after thousands of generations, had started having trouble metabolizing glucose and instead had started to metabolize citrate. This was a big deal, and was touted as an important evolutionary step for the E coli. As New Scientist put it,
"…But sometime around the 31,500th generation, something dramatic happened in just one of the populations - the bacteria suddenly acquired the ability to metabolise citrate, a second nutrient in their culture medium that E. coli normally cannot use."
To the common reader, that sounds as though E coli mutated a brand new trait out of thin air. Especially since New Scientist goes on to say, "Indeed the inability to use citrate is one of the traits by which bacteriologists distinguish E. Coli from other species."
It is true that regular old E coli doesn't normally metabolize citrate. However, it does have the ability to do so under specific conditions. In August 1998, the Journal of Bacteriology published an article on the ability of E coli to convert citrate to acetate and succinate under anoxic conditions (the absence of oxygen) when an oxidizable cosubstrate like glucose is present. In other words, if sugar is present and oxygen isn't, E coli does have the capacity to "eat" citrate.
Discovering exactly what happened to "up" this ability is up to Dr. Lenski's team. He has samples of E coli populations from thousands of generations over the years, and he can pinpoint the specific changes that led to make E coli's already existing citrate carrier expand its horizons.
We just find it interesting that these citrate-munching E coli have also lost a lot of their ability to eat glucose, their normal food.
Evolutionists argue that evolutionary change doesn't always have to be a drive upward. They say that evolutionary change can offer benefits at the same time as losing other useful functions. That's fine. Except that we never see any examples of truly upward "change." If there is a new or improved ability in an organism, we find that it was always tucked away there in the genetic code. Otherwise, "new" traits tend to come with a loss of information, a loss of function, a mistake, an error that might temporarily offer some benefit to the creature at hand, but in the long run harms it. The man with no esophagus will have a hard time getting sick from a foodborne illness, but few people will argue that living by feeding tube is a long-term beneficial "adaption." Evolutionists keep trying to argue that similar losses or defects offer beneficial adaption, but all we see in these mutations is net deterioration.
In all this, we find that bacteria are still bacteria. They are not developing new organelles that were not previously present. For good or bad, fully functioning or not, they just continue to behave like bacteria. We say, aside from thousands of years of genetic weakening, they are still doing what God designed them to do.
November 9, 2009 9:30 a.m. to 5:00 p.m. St. Pius V University (Rome)
The 150th anniversary of Darwin’s "Origin of the Species" in November 2009 will be the occasion for a unique conference at Pope Pius V University in Rome presenting a scientific refutation of evolution theory. According to Russian sedimentologist Alexander Lalamov, “Everything contained in Darwin’s Origin of Species depends upon rocks forming slowly over enormous periods of time. The November conference demonstrates with empirical data that such geological time is not available for evolution.” Recently returned from a ground-breaking geological conference in Kazan, sedimentologist Guy Berthault will present the findings of several sedimentological studies conducted and published in Russia. In one of these, the age of the rock formation surveyed was found to be 0.01% of the age attributed to it by the geological time-scale—instead of an age of 10 million years, the actual age was no more than 10 thousand years. “Contrary to the conventional wisdom,” Lalamov observed, “these rocks formed quickly, and the fossils they contain must be relatively young. This finding contradicts the evolutionary interpretation of the fossil record.” www.sedimentology.fr
According to US biophysicist Dr. Dean Kenyon, “Biological macroevolution collapses without the twin pillars of the geological time-scale and the fossil record as currently interpreted. Few scientists would contest this statement. This is why the upcoming conference concentrates on geology and paleontology. Recent research in these two disciplines adds powerful support to the already formidable case against teaching Darwinian macroevolution as if it were proven fact.”
Participating scientists include:
--Guy Berthault, a renowned sedimentologist from France and experimenter in fundamental physics and sedimentology, member of the French Geological Society and the Association of Sedimentologists.
--Maciej Giertych, a population geneticist from Kornik, Poland, who holds advanced degrees in genetics, forestry and tree physiology.
--Thomas Seiler, a physicist from Germany with a Ph.D. in physics from the University of Munich
--Jean de Pontcharra, a physicist in France and director of the renowned research group CEA-LETI (Commissariat à l'Energie Atomique, Laboratoire d'Electronique et de Technologie de l'Informatique).
--Josef Holzschuh, a geophysicist from Australia with a Ph.D. in geophysics from the University of Western Australia.
Abstracts of the presentations can be seen on-line at http://sites.google.com/site/scientificc
“The Scientific Impossibility of Evolution” conference is being held in direct response to Benedict XIV's request that both sides of the evolution controversy be heard. Thomas Seiler, a participant in the conference said: “In the light of astounding new scientific breakthroughs, particularly in geology, we hope the worldwide scientific community will acknowledge the overwhelming evidence against the theory of evolution.”
The conference begins at 9:30 a.m. on November 9 in the auditorium of St. Pius V University (Via Cristoforo Colombo, No. 200). Entrance is free although the number of seats is limited. It is open to the public and members of the press and media, but reservations are recommended. Reservations can be made by email at noevolutioninfo@gmail.com; or Peter Wilders (Europe phone 377 93 50 88 34)
The field of biology has provided much support for a recent creation, and physical evidence of very young-looking biological materials from supposedly ancient fossils continues to accrue from around the world, and from various depths under the earth.
In August of this year, paleontologists in Trowbridge, Wiltshire, England, made a discovery that astounded the evolutionary community. A "150 million-year-old" squid was discovered with an intact ink sac. "It is difficult to imagine how you can have something as soft and sloppy as an ink sac…inside a rock that is 150 million years old," said Dr. Phil Wilby of the British Geological Survey.1 Creationists agree and see this as physical evidence that clearly points to its recent burial and preservation.
"Living fossils" present another kind of dilemma for deep time. Sharks, horseshoe crabs, crinoids, Wollemi pine trees, tuataras, crocodiles, vampire squids, chambered nautiloids, brachiopods, clams, dragonflies, lungfish, and hundreds of other animals and plants have stayed the same over "millions of years," despite significant shifts in their environments and supposed eons of nature-selecting mutations. But living fossils make better sense when viewed from a young-earth creation perspective, wherein natural selection does not generate new forms, and living creatures ought to look similar to their fossilized relatives.
In the past few decades, Darwin-unfriendly discoveries have been made of genetic material (DNA) that is supposedly from very old sources but is found in relatively pristine condition. For example, evolutionist Sangtae Kim discovered DNA sequences from Miocene fossils (supposedly over 5 million years old) and said, "This paper confirms that DNA sequences can be obtained from Miocene-age plant remains."2 And "plants, bacteria, mammals, Neanderthals, and other archaic humans have had short aDNA [ancient DNA] sequences identified."3 How can DNA be so intact after so long, with what is known about DNA decay rates?4, 5
Other samples of viable DNA have been extracted from frozen tissue dated thousands of years old by evolutionists. The energy powerhouse of the cell is called the mitochondria and it contains DNA termed mtDNA. In 2008, samples of mtDNA were extracted from a frozen human discovered in the Alps in 1991. Called the Tyrolean Iceman, he was dated at over 5,000 years old. The DNA was completely and successfully sequenced.6
Speaking of cold temperatures, deep ice cores have been taken from southern Greenland permafrost. Insect and plant materials recovered from them have revealed clean DNA sequences.7 Antarctic ice cores may reveal more DNA samples.
Not only is DNA found where it should not be if evolutionary ages are true, but still-living microbes have been extracted from ancient earth materials. A leader in this fascinating field is evolutionist Raul Cano of the California Polytechnic State University. His work frustrates evolutionary biologists, who maintain that the earth is very old, and therefore consider that his otherwise astonishing results are actually contaminated with recent bacteria. However, other evolutionists have been making similar discoveries.8
It would seem that many scientists are putting the cart before the horse--embracing long ages before they consider the physical evidence that shows otherwise.9 Pristine DNA from these supposedly ancient materials is predicted by the creation model, which numbers the earth's years in the thousands rather than millions.
References
* Mr. Sherwin is Senior Science Lecturer at the Institute for Creation Research.
Cite this article: Sherwin, F. 2009. Squid Fossils, Ancient DNA, and a Young Earth. Acts & Facts. 38 (10): 16.
Evolutionists have maintained that the fossil record supports a long-ages history for earth, but material extracted from dinosaur bones is providing an interesting challenge to that theory. The recent discoveries of soft dinosaur tissues, defined cell matrices, elastic blood vessels, and clearly observable cell microstructures such as cell nuclei have been a source of both shock and excitement to the paleontology community.
The shock comes from the fact that degradative processes somehow did not completely destroy all evidence of tissue from the supposedly millions-of-years-old fossils. The excitement comes from the fact that, given the pristine state of these tissues, scientists should be able to extract macromolecules. These would then be used in studies of molecular evolution to bolster the evolutionary ideas that are competing for supremacy in the scientific community, such as the currently touted "dinosaur to bird" transition model.
In fact, soft tissues from the bones of a Tyrannosaurus rex and a Brachylophosaurus canadensis (duck-billed hadrosaur) did yield protein fragments that were subjected to amino acid sequence analysis and then used in theoretical computational analyses.1, 2 But did the data demonstrate a dinosaur to bird transition, or was it possibly manipulated in the spirit of academic politics?
The First Protein Sequences
A protein is a chain of amino acids and, generally speaking, is the functional end-product of a gene. Evolutionary scientists commonly use both DNA and protein sequences in comparative analyses, comparing the same type of gene or protein sequence between organisms to determine how closely related one is to another. Two organisms are considered closely related if they share a high percentage of amino acid sequence similarity for a certain protein. Evolutionary tree diagrams can be constructed based on this concept of sequence similarity, with the branches and grouping of organisms supposedly indicating their evolutionary relationships.
As things stand, the dinosaur proteins that were characterized are largely controlled by the dinosaur-to-bird proponents. Jack Horner, a world-renowned paleontologist, is a leading figure in this group. His faith in the dino-to-bird concept is so strong that he recently published a book describing how one might possibly reverse-engineer a dinosaur by modifying key developmental genes in the chicken genome.3 Dr. Mary Schweitzer, one of his colleagues and his former graduate student, is the leading scientist in the United States working with dinosaur soft tissue. Dr. Schweitzer and protein biochemist John Asara have led the effort to research and publish the dinosaur protein sequence findings.
The first protein sequences to be characterized and analyzed were collagen proteins from a T. rex femur bone, in which a number of papers were published describing both the soft-tissue and protein data.4,5,6,7 Collagen is a very durable protein that is common to most animals and is found in skin, bone, and other connective tissue.
In general, the scientific community found very little to dispute regarding the presence of real dinosaur tissue such as blood vessels and intact bone matrix, clearly defined cell types, and clearly defined cell microstructures such as nuclei and filipodia (osteocyte tendrils). The recently published T. rex collagen sequences, however, have met with some legitimate criticism from scientists who specialize in protein characterization and analysis techniques.
Protein Sequence Methodology
In order to understand their criticisms, it is important to know something of protein sequencing methodology. First, proteins are isolated and separated into subgroups based on their various masses. Then they are chopped into small fragments using an enzyme called trypsin. The trypsinized fragments are then run through a highly specialized instrument called a mass spectrometer, which determines the mass of each protein fragment (peptide) in the sample.
A tandem mass spectrometer setup will not only determine the mass of the trypsin fragments in the initial sample, but will also send them through a second mode where they are physically fragmented further and the mass of these sub-fragments is also determined. Peptide mass databases are then searched for matching fragment sizes for all data collected. Using a specialized algorithm on the sub-fragment data, it is possible to computationally assemble the actual amino acid sequence of a sizeable peptide fragment.
The first accusation against the T. rex protein sequences was that they were too small and had possibly suffered too many chemical modifications to be reliable.8, 9 It was also pointed out by critics that the lab that published the protein sequence data did not indicate if or how they controlled error rates, such as the discovery of false positives.9 Establishing the proper experimental controls and statistical measures for the presence of false positives is essential to providing an accurate protein sequence, especially for ancient proteins. As one journal article that critiqued the protein data stated, "Extraordinary science requires extraordinary proofs."9
A Dino-to-Bird Filter
Based on reported experimental methods and deduced peptide sequences, error rates would have been unacceptably high for all but one of the sequences the researchers reported. By ignoring error rates, one could choose from among hundreds of peptide fragments in a database those that most closely resemble bird proteins. In fact, a number of dinosaur protein fragments were chosen with 100 percent amino acid similarity to that of chicken collagen. One published critique quipped, "Maybe T. rex was a chicken after all."9
Those sequences of high enough quality to be usable were then analyzed using dino-to-bird evolution as a filter.10 Interestingly, an external laboratory re-analyzed the data using a computational technique called Neighbor-Net analysis that was better suited to the type of data collected.8 Their results showed that the T. rex protein grouped more closely with amphibians and did not show a close relationship with either chicken or ostrich--two birds that evolutionists like Jack Horner claim actually have dinosaur genomes with just a few minor differences to make them birds.
Having said all that, there is no doubt that fragments of real dinosaur proteins were obtained, because antibody experiments conclusively identified collagen in the tissue samples. The problem is that the quality of the samples was very poor, fragment identification did not properly account for error, and the evolutionary analyses appear to have been manipulated to support a politically correct dinosaur-to-bird model.
The more recent hadrosaur collagen sequencing appears to have been handled with more care in the lab side of the project, and peptide sequences of much larger size were reported and submitted to the public databases.2 So far, there has not been much time for critical responses to have been published, but it appears that the ancient protein recovery and sequencing techniques have improved. However, once again the dinosaur sequences are represented as being closer to chicken and ostrich than even other reptiles.
Where is the Data?
At the Institute for Creation Research, a number of preliminary protein alignments have been done using different algorithms at a variety of alignment/gap parameter settings. In these studies, the large T. rex peptide fragment and the hadrosaur protein sequences typically align more closely with a variety of animals other than chicken. The ostrich sequence was generated in-house by the Schweitzer-Asara group and, rather oddly, has never been submitted to any of the public protein database repositories. This is also the case with the alligator collagen sequence they developed in-house.
At the time of this article, DNA/protein database searches at both the National Center for Biotechnology Information and the European Molecular Biology Laboratory have contained no alligator or ostrich collagen sequence. While it is possible to obtain the ostrich and alligator sequence data from material on the Internet posted as supplements to publications, why has the data not been submitted to any of the major public databases so it can be cataloged, annotated, and curated? This seems a little odd, considering that the researchers readily submitted all of the possibly errant T. rex sequence to the public databases.
Based on comments about hypothetical sequences being utilized during the procurement of the ostrich data (which also included real mass-spec data), how does one know if the ostrich sequence wasn't manipulated in the process to be more dinosaur-like? The authors do state that the hypothetical ostrich sequence developed was based on a dino-to-bird transitional model.1
Conclusion
Although the supposedly 90 million-year-old hadrosaur collagen sequence appears to have been interpreted within the assumption of dino-to-bird evolution--a concept that a number of other leading evolutionists do not share--the fact that real tissue and proteins have been found seriously brings into question the whole concept of evolution and its required long ages. The remarkable preservation of these tissues found in sedimentary rock (sandstone) really speaks of only one thing: a rapid burial in a catastrophic worldwide flood as recorded in the Bible.
In fact, even evolutionists have contemplated the implications, as illustrated in the quote below from Jack Horner's recent book. The setting for this excerpt is a conversation between Dr. Horner and Mary Schweitzer when she was his graduate student. Schweitzer had just discovered and verified the presence of intact dinosaur tissue and was relaying the news to her mentor.
When Mary was first working on this material, she called me up to say she had found osteocytes. I assumed she meant the spaces where the osteocytes would have been, which is what I suggested.
"No, Jack, actually we have the cells and they have filipodia and they have nuclei."
"Mary, the freaking creationists are just going to love you."
"Jack, it's your dinosaur."11
That about sums it up!
References
* Dr. Tomkins is Research Associate at the Institute for Creation Research.
Cite this article: Tomkins, J. 2009. Dinosaur Protein Sequences and the Dino-to-Bird Model. Acts & Facts. 38 (10): 12-14.
Dinosaurs are a popular topic of study, whether in the public imagination or in scientific research. The scientific community, however, has a dirty little secret regarding the manner in which that research is handled. If dinosaur DNA doesn't "look like chicken" (or a crocodile), it will most likely be discarded as "unreliable data" prior to publication--and thus be effectively censored from public access.
Why? Because evolutionary scientists are committed to only publish dinosaur DNA data that match their naturalistic tale of origins. Despite the amazing discoveries of soft tissue from dinosaur bones,1 dinosaur DNA research results (and other dinosaur "connective tissue" research) continue to be steered by evolutionary dogmatism.
Dino DNA
An article published in Science in 1993 illustrates how and why dinosaur bone research has been chillingly censored. "Dino DNA: The Hunt and the Hype" by Virginia Morell stated that "several groups are racing to get the first DNA out of dinosaur bones, but other researchers say their efforts are taking attention away from the real scientific value of ancient DNA."
This article referenced then-recent findings of fresh dinosaur tissue:
Mary Schweitzer, a biology graduate student at Montana State University's Museum of the Rockies, was examining a thin section of Tyrranosaurus rex bone…when she noticed a series of peculiar structures. Round and tiny and nucleated, they were threaded through the bone like red blood cells in blood vessels. But blood cells in a dinosaur bone should have disappeared eons ago. "I got goose bumps," recalls Schweitzer. "It was exactly like looking at a slice of modern bone. But, of course, I couldn't believe it. I said to the lab technician: 'The bones, after all, are 65 million years old. How could blood cells survive that long?'"2
Why was Schweitzer, an eyewitness who microscopically observed the insides of a T. rex bone, afraid to believe her own eyes? Isn't empirical science all about observation? Furthermore, Morell reported, "Schweitzer has already extracted a molecule that might be dinosaur DNA."
However, connective tissue ruins and degrades over time, such that DNA should not survive at all, even if the creature only lived 50,000 years ago.3 The existence of 65 million-year-old DNA is biochemically unthinkable. In other words, the old-earth evolutionary tale is clearly at odds with the fresh dinosaur bone evidence. How embarrassing to the academic establishment! This may be why ongoing dinosaur soft tissue discoveries are generally not broadcast through popular media channels.
Research Censorship
Evolutionary "damage control" is observed in the form of "chilling" (i.e., coerced) censorship of research, with severe consequences to those who "buck the system." Consider the research flow chart pictured below describing the process of extracting dinosaur DNA. Note steps 7 and especially 8. Why must the research results be dismissed if the DNA extract doesn't look like birds or crocodiles? The answer is evolutionary gatekeeping:
To make sure she's liberated the right molecule, Schweitzer compares the extracted DNA sequences with those of hundreds of living organisms. If the sequence turns out to be similar to that of a known fungal gene, for example, she knows the sample has been contaminated.
That's how DNA hunters know they've gone wrong. But how do they know when they're on the right track, given that there are no living dinosaurs to provide a handy sample of DNA for comparison? The answer is that they rely on paleontological theory, which (according to most researchers) holds that dinosaurs and crocodiles came from the same stock, and that the dinosaurs' only living descendants are birds. Therefore researchers look for DNA that is similar, but not identical, to DNA from these groups of organisms.4
In other words, only DNA research that provides dinosaur DNA sequences similar to those of birds and crocodiles is allowed. As the flowchart indicates, all other results are deemed anomalies that should be rejected as though they were known contaminants, like fungal genes. This approach is not observation-directed empirical research; this is assumption-driven, theory-dictated censorship--"science" falsely so-called.5

Coerced Spoliation of Evidence
This purposeful pattern of coerced concealment of the nonconforming DNA data from unfossilized dinosaur bones (labeled "an anomaly" on the chart) involves what courtroom lawyers and judges call "chilling" coercion and "spoliation of evidence"--inducing the concealment (and eventual destruction) of embarrassing information in order to prevent one's opponent from using it at trial.
Whenever any kind of evidence is concealed, one immediately questions the spoliators' motives for doing so. The intuitive answer is that they dislike what the information would reveal. Therefore, to spoliate evidence suggests that the spoliators' argument or theory would be weakened, or embarrassed, by that evidence. This suggestion is so strong, forensically speaking, that it is treated as a rule of presumptive inference in law courts. In other words, if someone hides evidence in this way, the law presumes that the hidden evidence was damaging to the argument of the spoliator. The spoliator then bears the burden of proof to show otherwise.6
A kindred rule to the foregoing…is that the intentional spoliation or destruction of evidence relevant to a case raises a presumption that the evidence would have been unfavorable to the cause of the spoliator.…The deliberate destruction of evidence gives rise to the presumption that the matter destroyed is not favorable to the spoliator.7
This shows that the civil law courts understand the importance of evidence spoliation--it points to a willingness to conceal or otherwise suppress truth in order to advance a specific cause. The name Arthur Andersen comes to mind, as this accounting firm's shredding of Enron documents hindered SEC investigators.8
Follow the Procedure, or Else
In suppressed dinosaur DNA research--which is a subset of the irrefutable, but hushed, dinosaur soft tissue discoveries--the same issue of evidence spoliation is relevant. Why? Because today's dinosaur DNA controversy in particular, and today's dinosaur "connective tissue" controversy in general, directly puts at issue the real age of the dinosaurs: Did they live millions of years ago, or in much more recent history on an earth inhabited by humans--descendants of Adam and Eve?9
How will anyone really know what dinosaur DNA sequences look like until uncensored data from dinosaur bones are published for public scrutiny? And how will such data be published at all if "embarrassing" research results are routinely discarded as anomalous, simply because they didn't "look like chicken"? One way to acquire more reliable data in this case would be to repeat the DNA research across multiple labs, until consistent results emerge.
In fact, a similar approach was taken in 1994. The winners of the race to sequence dinosaur DNA were Scott Woodward and his colleagues, who published their results in Science.10 They extracted DNA from a purportedly well-preserved dinosaur bone. However, they were not rewarded for their victory. The sequence they discovered was not like birds or reptiles, but seemed unique.
These researchers decided not to follow the procedure outlined in the 1993 flowchart, which would have "told" them that what they found was an unacceptable "anomaly." Since this 1994 DNA did not fit the evolutionary interpretive filter, the authors were raked over the academic coals. Moreover, the objections to their results were not based on conflicting research results, but appeared in editorials and reviews. As a result of the uproar from the scientific community, their dinosaur DNA sequence never became a permanent entry in any public database. In fact, since this very public academic flogging, no scientist has attempted to publish any dinosaur DNA research (resulting in "chilled" academic speech).
Interestingly, Schweitzer has never published any of her purported DNA research on dinosaur tissue, although she has published on tissue analyses and, recently, data on protein sequence. While the tissue analyses reported over the past decade are nearly impossible to dispute, this recently published dinosaur protein sequence from a T. rex came under extreme criticism and the data were highly questioned by peers as having been manipulated to produce close similarities with chicken and ostrich protein.11 Was this done as per the "paleontological theory and protocol" described in 1993?
Conclusion
The gatekeeping approach to ancient DNA research established as a protocol in 1993 is a product of dogmatic evolutionary theory. The 1994 results put the dogma to the test, with the result that:
The coerced suppression of the results by the evolutionary scientific community has dissuaded anyone else from publishing dinosaur DNA research that is not in line with evolutionary dictates. Such self-censorship "chills" empirical research, which prevents the public reporting of observable DNA sequences in order to insulate the larger story of particles-to-people evolution from cross-examination.
Where are the real scientists in dinosaur DNA research who refuse to kowtow to evolution's gatekeepers?
References
* Dr. Johnson is Special Counsel at ICR. Dr. Tomkins, ICR Research Associate, worked in academic research in genetics and genomics for 18+ years, 12 involving research in cloning and sequencing DNA from a wide variety of plants, animals and microbes. Mr. Thomas is Science Writer at the Institute for Creation Research.
Cite this article: Johnson, J. J. S., J. Tomkins and B. Thomas. 2009. Dinosaur DNA Research: Is the tale wagging the evidence? Acts & Facts. 38 (10): 4-6.
Dinosaur Soft Tissue Issue Is Here to Stay
by Brian Thomas, M.S.*
In recent decades, soft, squishy tissues have been discovered inside fossilized dinosaur bones. They seem so fresh that it appears as though the bodies were buried only a few thousand years ago.
Since many think of a fossil as having had the original bone material replaced by minerals, the presence of actual bone--let alone pliable blood vessels, red blood cells, and proteins inside the bone--is quite extraordinary. These finds also present a dilemma. Given the fact that organic materials like blood vessels and blood cells rot, and the rates at which certain proteins decay, how could these soft tissues have been preserved for ten thousand, let alone 65 million or more, years?
These soft tissues have met with hard resistance from mainstream science, and some scientists have even discounted or ignored them. But fresh studies keep finding fresh tissue, making the issue difficult to dismiss. Either the vast evolutionary ages assigned to these finds are dramatically erroneous, or "we really don't understand decay" rates of the soft tissues and proteins.1
Paleontologists who have analyzed the tissues, visible through their microscopes and squeezable with their tweezers, insist that something is fundamentally wrong with laboratory data on biochemical decay rates.2 In turn, biochemists are confident that their repeatable experiments show that the soft tissues should not be there after all this time. To try to get around the hard facts of soft tissues, some scientists have even proposed that the blood vessels and red blood cells in question were bacterial slime. This was thoroughly refuted, however, by research showing that the dinosaur tissue contains a collagen protein that bacteria do not produce.3
This dilemma between the science of biochemistry and the belief in millions of years is not going away. In addition to the well-characterized tissues from a T. rex reported by paleontologist Mary Schweitzer in 1997,4 2005,5 and 2007,6 new soft tissue finds keep surfacing. Schweitzer published a report on another sample in Science in 2009,3 this time from a hadrosaur, in which the precise characteristics of dinosaur biochemicals were verified by a third party. This was necessary to confirm the reality of the soft tissues to an incredulous scientific community. (Similarly, Schweitzer's 2007 results have also been verified.7)
Yet another hadrosaur has been described by UK scientists as "absolutely gobsmacking."8 Its tissues were "extremely well preserved" and contained "soft-tissue replacement structures and associated organic compounds."9
Schweitzer's team recently concluded that "the most parsimonious explanation, thus far unfalsified, is that original molecules persist in some Cretaceous dinosaur fossils."3 But biochemical decay rates showing that soft tissues would be dust after all this time are also thus far unfalsified (i.e., have not been disproved). Therefore, the millions-of-years age assignments must go.
However, if the deep time goes, then so does the grand story of evolution that depends on it. For many, that is too sacred an assumption to dare alter. Biblical data, however, not only provide the timeframe for the death of these dinosaurs in Flood deposits a few thousand years ago, but also a mode of deposition in agreement with observable data that their demise occurred when they "fell into a watery grave."8
References
1. Fields, H. 2006. Dinosaur Shocker. Smithsonian magazine online. Published May 2006, accessed July 20, 2009.
2. For example, see Bada, J. L., X. S. Wang and H. Hamilton. 1999. Preservation of key biomolecules in the fossil record: current knowledge and future challenges. Philosophical Transactions of the Royal Society B. 354 (1379): 77-87.
3. Schweitzer, M. H. et al. 2009. Biomolecular Characterization and Protein Sequences of the Campanian Hadrosaur B. canadensis. Science. 324 (5927): 626-631.
4. Schweitzer, M. and T. Staedter. 1997. The Real Jurassic Park. Earth. 6 (3): 55-57.
5. Schweitzer, M. et al. 2005. Soft-Tissue Vessels and Cellular Preservation in Tyrannosaurus rex. Science. 307 (5717): 1952.
6. Asara, J. M. et al. 2007. Protein Sequences from Mastodon and Tyrannosaurus Rex Revealed by Mass Spectrometry. Science. 316 (5822): 280-285.
7. Bern, M., B. S. Phinney and D. Goldberg. 2009. Reanalysis of Tyrannosaurus rex Mass Spectra. Journal of Proteome Research. Published online July 15, 2009.
8. Mummified dinosaur skin yields up new secrets. The University of Manchester press release, July 1, 2009.
9. Manning, P. L. et al. 2009. Mineralized soft-tissue structure and chemistry in a mummified hadrosaur from the Hell Creek Formation, North Dakota (USA). Proceedings of the Royal Society B. Published online before print, July 1, 2009.
Image Credit: Christian Darkin / Photo Researchers, Inc.
* Mr. Thomas is Science Writer at the Institute for Creation Research.
Cite this article: Thomas, B. 2009. Dinosaur Soft Tissue Issue Is Here to Stay. Acts & Facts. 38 (9): 18.